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Dupixent Wrongful Death Claims

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When CTCL Becomes Fatal: The Richardson Case and Beyond

Cutaneous T-cell lymphoma is an indolent disease in its early stages, but advanced CTCL — particularly large-cell transformation, Sezary syndrome, and visceral involvement — carries high mortality. Stage IVB CTCL has a median survival of approximately 1 to 2 years. When a Dupixent patient's CTCL progresses to these advanced stages and the patient dies, the litigation shifts from personal injury to wrongful death and estate claims.

Richardson v. Sanofi US Inc. et al., filed in the Western District of Tennessee in October 2025, was among the earliest named Dupixent CTCL lawsuits nationally. The case brought public attention to the fatal trajectory that Dupixent-associated CTCL can follow: a patient treated for atopic dermatitis who developed lymphoma during treatment and experienced rapid disease progression. The Richardson filing catalyzed additional lawsuits and contributed to the JPML petition for MDL consolidation.

Fatal CTCL cases carry the highest damage potential in Dupixent litigation. Juries are confronted with a patient who sought relief from a skin condition and instead received a death sentence from a cancer that the manufacturer knew — or should have known — was a risk of its product. The emotional impact of these cases during bellwether trial selection makes wrongful death claims particularly valuable to plaintiffs' steering committees in MDL proceedings.

Wrongful Death vs. Survival Action: Two Separate Claims

A wrongful death claim is brought by the decedent's surviving family members (or a personal representative on their behalf) and compensates the survivors for their own losses: loss of the decedent's companionship, guidance, and consortium; loss of the decedent's financial contributions and earning capacity; and funeral and burial expenses. The claim belongs to the survivors, not to the estate, and the damages reflect the impact of the death on the living.

A survival action, by contrast, is brought by the decedent's estate and recovers damages that the patient could have recovered had they survived: pain and suffering from diagnosis through death, medical expenses incurred during treatment, lost wages during illness, and emotional distress. The survival action essentially 'survives' the decedent's death and passes to the estate. In most jurisdictions, both claims can be pursued simultaneously, and the combined damages from a wrongful death claim plus survival action can significantly exceed a living plaintiff's personal injury recovery.

Who Has Standing to File

Wrongful death standing is controlled entirely by state law, and the rules vary dramatically. In most states, a surviving spouse and minor children have automatic standing. In some states, adult children, parents, and even siblings or domestic partners may also bring wrongful death claims. A handful of states restrict standing to the personal representative of the estate, who files on behalf of all statutory beneficiaries. In states like Alabama, the wrongful death claim is punitive only — it compensates nothing for the survivors' loss but instead punishes the defendant, with the entire recovery going to the estate.

For Dupixent wrongful death claims, identifying the correct state law is critical. The applicable law is typically the state where the death occurred or the state where the decedent was domiciled. If the patient was treated in one state, moved to another for hospice care, and died in a third state, choice-of-law analysis may determine which state's wrongful death statute applies. An attorney experienced in pharmaceutical wrongful death must evaluate standing, applicable law, and filing deadlines before the relevant statute of limitations expires.

State Statute of Limitations Variations for Wrongful Death

Wrongful death statutes of limitations are separate from personal injury SOLs and are typically measured from the date of death, not from the date of diagnosis. Tennessee's wrongful death SOL is 1 year from the date of death — the shortest in the nation. California allows 2 years. New York allows 2 years. Illinois allows 2 years under the Wrongful Death Act but 1 year under the Survival Act if the decedent had not filed suit before death. Florida allows 2 years. Pennsylvania allows 2 years. These tight deadlines mean that families grieving a Dupixent-related death have limited time to consult counsel and file suit.

The discovery rule may extend these deadlines in some states if the family did not know — and could not reasonably have known — that Dupixent caused or contributed to the CTCL that led to death. However, as Dupixent CTCL litigation gains media attention and MDL proceedings publicize the risk, courts may be less willing to apply the discovery rule to new claimants. Families should not assume they have time; the safest course is to consult a pharmaceutical liability attorney within months of a Dupixent patient's death from CTCL.

Damages in Fatal Dupixent CTCL Cases

Damages in fatal CTCL cases encompass multiple categories. The survival action component includes the patient's pre-death pain and suffering — which in advanced CTCL can involve total-body erythroderma, severe pruritus, repeated hospitalizations, chemotherapy, radiation, and stem cell transplantation — plus all medical costs from diagnosis through death. The wrongful death component includes loss of financial support (often calculated using vocational economists who project lifetime earnings), loss of household services, loss of parental guidance for minor children, loss of consortium for a surviving spouse, and funeral expenses.

Punitive damages are available in both wrongful death and survival actions in most jurisdictions, and the fatal nature of the injury supports a strong punitive case. Evidence that Sanofi and Regeneron knew of the CTCL signal, failed to warn, and continued aggressive marketing to expand Dupixent's patient base supports a finding of the reckless disregard or conscious indifference required for punitive awards. Combined wrongful death and survival action damages in fatal pharmaceutical cases routinely reach eight figures in jury verdicts.

FAQ

Frequently Asked Questions

Can Dupixent cause cancer?

The short answer is yes — and the science behind it is alarming. A peer-reviewed study of 19,612 patients found that Dupixent users face a 4.5 times higher risk of developing cutaneous T-cell lymphoma (CTCL) compared to people who never took the drug. To put that in perspective, a 4.5x relative risk is in the same ballpark as the asbestos-mesothelioma association that drove one of the largest mass tort litigations in American history. The proposed biological mechanism is straightforward and troubling. Dupixent blocks two signaling molecules — IL-4 and IL-13 — that are part of the type 2 immune response. Blocking these signals is what makes the drug effective against eczema and asthma. But those same signals appear to play a role in immune surveillance against T-cell malignancies. By suppressing them, Dupixent may release the brakes on pre-malignant T-cell clones that the immune system had been keeping in check. The result: a cancer that literally disguises itself as the disease the drug is supposed to treat. The FDA placed Dupixent on its safety watchlist in March 2025 and escalated to a formal investigation in September 2025 after receiving more than 300 adverse event reports related to lymphoma and blood cancers. As of April 2026, the investigation is ongoing. Regeneron and Sanofi have not added a specific CTCL warning to the Dupixent label.

What is cutaneous T-cell lymphoma, and why is it connected to Dupixent?

Cutaneous T-cell lymphoma is a cancer of the immune system that begins in the skin. Specifically, T-lymphocytes — white blood cells that are supposed to protect you from infections and abnormal cells — become malignant and accumulate in the skin, forming patches, plaques, and eventually tumors. The most common subtype is mycosis fungoides, which can remain indolent for years or progress to Sezary syndrome, an aggressive form where cancer cells circulate through the bloodstream. The connection to Dupixent lies in the drug's mechanism of action. Dupixent works by blocking IL-4 and IL-13 — two signaling molecules that drive type 2 inflammation. But these molecules also appear to play a role in keeping abnormal T-cells under control. When Dupixent shuts down that pathway, it may allow pre-existing T-cell clones with malignant potential to proliferate unchecked. Researchers describe this as an 'unmasking' effect — the cancer was there, but the immune system was holding it back. Dupixent may remove that restraint. The most dangerous aspect of this connection is diagnostic mimicry. Early CTCL looks almost identical to eczema — red, scaly patches on the skin. For a patient taking Dupixent for eczema, new skin lesions are almost always attributed to eczema flares rather than cancer. This can delay diagnosis by months or years, allowing the cancer to progress to stages where treatment options narrow and survival rates drop sharply.

Who qualifies to file a Dupixent lawsuit?

If you took Dupixent for any condition — atopic dermatitis, asthma, chronic sinusitis, COPD, or any other indication — and were subsequently diagnosed with cutaneous T-cell lymphoma (including mycosis fungoides or Sezary syndrome), you may have a valid claim. There is no minimum duration of Dupixent use required. Some reported cases involve patients who developed CTCL after relatively short treatment periods, which is consistent with the unmasking theory: if Dupixent releases the brakes on a pre-existing malignancy, the cancer can manifest quickly. Family members of people who died from CTCL or T-cell lymphoma after Dupixent use may be eligible to file wrongful death claims. You do not need to have stopped taking Dupixent — active patients with a CTCL diagnosis can file while still receiving treatment for their cancer. The key requirements are documentation of Dupixent use (pharmacy records, insurance claims, or medical records), a confirmed CTCL or T-cell lymphoma diagnosis (pathology report with biopsy confirmation), and filing within your state's statute of limitations. A free consultation with a mass tort attorney can determine whether your specific circumstances support a claim. Consultations are confidential, and most Dupixent attorneys work on contingency — fee terms vary by attorney.

What is the current status of Dupixent lawsuits in 2026?

As of April 2026, all federal Dupixent lawsuits alleging cancer injuries have been consolidated into Multidistrict Litigation No. 3180 by the Judicial Panel on Multidistrict Litigation. The MDL is in its earliest procedural stages — the court has appointed a plaintiffs' steering committee, and initial case management orders are being issued. Discovery has not yet begun in earnest, but attorneys expect the first round of document requests to target Regeneron's internal safety data, communications with the FDA, and pharmacovigilance records. No settlements have been reached and no cases have gone to trial. The litigation follows a well-established pattern for pharmaceutical MDLs: after discovery is substantially complete, the court will select bellwether cases for trial. Bellwether verdicts — typically the first three to five cases tried — establish the litigation's settlement value and create pressure for global resolution. This process typically takes two to four years from MDL formation, meaning bellwether trials could begin in 2028 or 2029. Separately, the FDA's formal investigation into Dupixent's cancer risk remains ongoing. Any regulatory action — particularly a boxed warning or label change requiring specific CTCL risk disclosure — would significantly strengthen plaintiffs' cases and could accelerate settlement discussions.

How much could a Dupixent lawsuit be worth?

It is too early to provide specific settlement projections for Dupixent cases because no settlements or verdicts have been reached. But we can look at comparable pharmaceutical cancer litigations to understand the range of possibilities. In the Roundup litigation, individual settlements for non-Hodgkin lymphoma ranged from approximately $100,000 to over $2 million depending on cancer severity and treatment burden. Bayer ultimately paid over $10 billion to resolve approximately 100,000 claims. For Dupixent, case values will likely be stratified by the severity of the CTCL diagnosis. Early-stage mycosis fungoides managed with skin-directed therapies may fall in the $75,000 to $200,000 range. Advanced CTCL requiring chemotherapy, radiation, or targeted biologic therapy could be valued at $200,000 to $1 million. Wrongful death cases and terminal diagnoses — particularly Sezary syndrome and large cell transformation — could exceed $2 million. One factor that may favor Dupixent plaintiffs is the strength of the epidemiological evidence. A 4.5x relative risk is considered highly significant in both medical research and legal causation standards. The Daubert challenge to plaintiffs' expert testimony — often the most contentious phase of pharmaceutical MDLs — may be less of an obstacle than in litigations with weaker statistical associations.

Did Regeneron and Sanofi know about the cancer risk?

This is the central question in Dupixent litigation — and the answer will emerge from discovery as the MDL progresses. What we know publicly is this: adverse event reports linking Dupixent to lymphoma and blood cancers had been accumulating in the FDA Adverse Event Reporting System (FAERS) for years before the formal investigation. The scientific literature on IL-4/IL-13 pathway disruption and immune surveillance against T-cell malignancies was available to Regeneron's and Sanofi's research scientists at the time of approval and throughout the post-market period. Plaintiffs allege that the manufacturers engaged in inadequate pharmacovigilance — that they failed to investigate the cancer signal with the urgency that the data warranted, and that they failed to update the drug's label to warn specifically about CTCL risk. The current Dupixent label mentions the possibility of malignancies in general terms, but does not specifically warn about cutaneous T-cell lymphoma or the diagnostic confusion that arises from prescribing an eczema drug that can cause a cancer mimicking eczema. If discovery reveals internal documents showing that Regeneron or Sanofi were aware of the CTCL signal and chose not to act — or that they deliberately framed CTCL reports as eczema treatment failures in their pharmacovigilance databases — it could dramatically increase both liability and damages, including potential punitive damages.

Can I file a Dupixent lawsuit if I am still taking the drug?

Yes — you do not need to stop taking Dupixent to file a lawsuit. If you have been diagnosed with CTCL or another T-cell lymphoma while using or after using Dupixent, your legal claim exists regardless of whether you continue the medication. The decision to stop or continue Dupixent is a medical decision that should be made with your treating physician, not your attorney. Some patients with severe atopic dermatitis or asthma have no adequate alternative treatments, and stopping Dupixent could cause significant health deterioration. That said, if you have a confirmed CTCL diagnosis, your oncologist will almost certainly recommend discontinuing Dupixent — continuing a drug that may be promoting T-cell malignancy while simultaneously undergoing cancer treatment is medically counterproductive. The important thing is to make this decision in consultation with your medical team, not based on legal strategy.

How is a Dupixent MDL different from a class action?

The distinction matters because it directly affects how much you can recover. In a class action, all members share a single settlement pot — often resulting in relatively small per-person payments. In a multidistrict litigation (MDL), each plaintiff maintains their own individual case with their own specific facts, damages, and potential recovery. The MDL structure simply consolidates the pretrial process — discovery, expert challenges, and procedural motions — before a single judge for efficiency. In MDL No. 3180, your case is uniquely yours. A patient diagnosed with early-stage mycosis fungoides managed with topical therapy will have a different case value than a patient who underwent chemotherapy for Sezary syndrome or whose family filed a wrongful death claim. The MDL process allows the court to handle common legal issues efficiently while preserving each plaintiff's right to individual assessment and compensation based on their specific circumstances. After bellwether trials establish settlement benchmarks, most MDLs move toward global resolution — a structured settlement program in which individual case values are assigned based on severity tiers, treatment burden, and other case-specific factors. Plaintiffs who are unsatisfied with their assigned value retain the right to opt out and pursue individual trial.

What evidence do I need to file a Dupixent lawsuit?

The evidence you need falls into two categories: proof of Dupixent use and proof of cancer diagnosis. For Dupixent use, the strongest evidence includes pharmacy dispensing records showing when you filled your prescriptions, insurance claims showing Dupixent charges, medical records with prescribing notes, and injection logs from your doctor's office or specialty pharmacy. Even partial records are useful — your attorney can subpoena pharmacy records and insurance claim histories to fill gaps. For your cancer diagnosis, the essential document is your pathology report from the skin biopsy that confirmed CTCL or mycosis fungoides. The report should include the histopathological findings, immunohistochemistry results (CD3+, CD4+, loss of CD7/CD26), and ideally T-cell receptor gene rearrangement results confirming clonality. Your oncology treatment records — including staging workups, PET scans, chemotherapy regimens, and radiation plans — document the severity and treatment burden that drive your case value. You do not need to have all of this documentation organized before consulting an attorney. Most mass tort firms have medical record retrieval teams that gather and organize evidence as part of case preparation. The most important step is to preserve what you have — do not discard Dupixent packaging, pharmacy receipts, or medical records.

My doctor says my CTCL is not related to Dupixent. Can I still file?

Yes — and this is more common than you might think. Many treating physicians are not yet aware of the epidemiological data linking Dupixent to CTCL, or they may be reluctant to attribute a rare cancer to a widely prescribed medication. Your doctor's clinical opinion is important for your medical care, but it is not determinative of your legal claim. In mass tort litigation, causation is established through expert testimony from epidemiologists, toxicologists, and oncologists who analyze population-level data and biological mechanisms. The 4.5x relative risk from the 19,612-patient study provides strong statistical support for general causation — the question of whether Dupixent can cause CTCL in the population. Specific causation — whether Dupixent caused your particular CTCL — is then established through expert analysis of your medical records, treatment timeline, and the absence of alternative explanations. An expert who reviews your chart and finds that you had no CTCL risk factors before Dupixent use, developed CTCL during or after treatment, and had a clinical presentation consistent with drug-induced T-cell lymphoma can provide a compelling specific causation opinion regardless of what your treating physician believes.

Should I stop taking Dupixent if I am worried about cancer?

This is a medical decision, not a legal one — and it should be made in consultation with your prescribing physician. Abruptly stopping Dupixent can cause severe rebound flares of atopic dermatitis or worsening of asthma that may require emergency treatment. Your doctor can evaluate your individual risk factors, discuss the emerging safety data, and consider whether alternative treatments are appropriate for your situation. What we do recommend is vigilance. If you are currently taking Dupixent, discuss the CTCL risk with your dermatologist and ask about a monitoring protocol. Any new or changing skin lesions — particularly patches that look different from your usual eczema, lesions in unusual locations like the buttocks or trunk, or skin changes that do not respond to standard eczema treatments — should prompt a biopsy rather than an assumption that it is just another flare. Early detection of CTCL, if it develops, dramatically improves outcomes. If you are diagnosed with CTCL, your oncologist will almost certainly recommend stopping Dupixent immediately. At that point, the medical and legal decisions align: you need cancer treatment, and continuing a drug suspected of promoting that cancer serves no purpose.

How long will the Dupixent lawsuit take to resolve?

Pharmaceutical MDLs are not quick — and anyone promising a fast resolution is not being honest. Based on the typical trajectory of comparable pharmaceutical cancer MDLs, the Dupixent litigation will likely follow this general timeline: 2026 to 2027 for discovery and expert development; 2027 to 2028 for Daubert challenges (where the court decides whether plaintiffs' and defendants' expert witnesses may testify); and 2028 to 2029 for bellwether trials — the first cases to go to trial. If bellwether verdicts favor plaintiffs, global settlement negotiations typically follow within 12 to 18 months. The realistic expectation for most plaintiffs is a resolution timeline of three to five years from the date of MDL formation — meaning 2029 to 2031 for the bulk of cases. Some cases may resolve sooner if early bellwether results are strongly in plaintiffs' favor and the defendants choose to negotiate rather than face repeated trial losses. The Roundup litigation, for comparison, produced its first plaintiff verdict in 2018 and Bayer announced a $10 billion settlement framework in 2020 — a two-year bellwether-to-settlement cycle. Early filing matters because cases filed early are most likely to be considered for bellwether selection, and bellwether plaintiffs often receive the highest individual recoveries. Filing early also ensures that your evidence is preserved, your medical records are secured, and your case is well-developed by the time settlement discussions begin.
Related Topics

Related Pages

CTCL Diagnosis After Dupixent Use

Cutaneous T-cell lymphoma is a rare and aggressive blood cancer that masquerades as eczema, psoriasis, and other benign skin conditions — making it uniquely dangerous for Dupixent patients who already carry dermatitis diagnoses. Misdiagnosis rates exceed 40 percent in early-stage CTCL because the symptoms overlap so precisely with the conditions Dupixent is prescribed to treat. Dermatologists expect itchy, red patches in their Dupixent patients; what they may not expect is that those patches now harbor malignant T-cells. Staging ranges from IA (limited patches) to IVB (visceral organ involvement and blood-borne Sezary cells), and the difference between a Stage IA diagnosis and a Stage IIB diagnosis can mean the difference between a normal life expectancy and a five-year survival rate below 40 percent. The critical diagnostic tool is a skin biopsy with immunohistochemistry, which can reveal hallmark Pautrier microabscesses — clusters of malignant T-cells in the epidermis that do not appear in benign dermatitis. Every Dupixent patient whose skin condition worsens, changes character, or fails to respond to continued treatment should demand a biopsy, not another refill.

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Dupixent Eye Problems and Ocular Side Effects

Conjunctivitis is the most common adverse event associated with Dupixent, occurring in approximately 30 percent of atopic dermatitis patients in clinical trials — a rate ten times higher than placebo. But conjunctivitis is just the visible surface of a broader ocular toxicity profile that includes blepharitis, keratitis, dry eye syndrome, limbal stem cell deficiency, and in rare but devastating cases, corneal perforation requiring emergency surgical intervention. These eye problems often develop within the first three months of treatment and can persist even after Dupixent is discontinued. Sanofi and Regeneron have acknowledged conjunctivitis on the Dupixent label but have minimized the severity spectrum, leading patients and prescribers to underestimate the risk of serious, vision-threatening complications. For patients who have suffered permanent vision impairment, corneal scarring, or required corneal transplantation due to Dupixent-related ocular injury, the manufacturer's characterization of these events as manageable side effects is contradicted by their clinical experience.

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Dupixent MDL Status and Litigation Update

The Judicial Panel on Multidistrict Litigation (JPML) is considering a petition to consolidate all federal Dupixent CTCL lawsuits into a single MDL for coordinated pretrial proceedings. As of early 2026, over 250 individual lawsuits have been filed across multiple federal districts, with plaintiffs proposing consolidation in the Northern District of Georgia (Atlanta) and defendants advocating for the Southern District of New York. MDL No. 3180 would centralize discovery, expert qualification (Daubert) proceedings, and bellwether trial selection under a single transferee judge. For individual plaintiffs, MDL consolidation means coordinated case management and potential leverage from shared discovery, but it also means potential delays as the court works through lead case selection and common issues. The venue decision — Atlanta versus New York versus other candidates — will shape the litigation's procedural landscape, judicial temperament, jury pool demographics, and ultimately the pressure on Sanofi and Regeneron to negotiate a global settlement.

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Dupixent Pediatric Use and CTCL Risk in Children

Sanofi and Regeneron have aggressively expanded Dupixent's pediatric indications, securing FDA approval for atopic dermatitis in children as young as 6 months old in 2022, following earlier approvals for ages 6 to 11 in 2020 and adolescents aged 12 to 17 in 2019. This expansion exposed millions of children with developing immune systems to a biologic drug that blocks IL-4 and IL-13 — cytokines critical to immune maturation, T-cell differentiation, and tumor surveillance. While CTCL is rare in children overall, the long-term consequences of sustained immune modulation during critical developmental windows are unknown because Sanofi's pediatric trials were not designed to detect rare cancers and follow-up periods were insufficient to capture latent malignancies. Pediatric Dupixent claims carry unique urgency: children have longer remaining lifespans over which to develop delayed-onset lymphoma, the immune insult occurs during a period of rapid lymphocyte development, and parents made treatment decisions based on safety assurances that did not account for the emerging CTCL signal. The duty to warn is heightened for pediatric drugs because parents — not the patient — make the consent decision, and they deserve complete risk information to exercise informed judgment.

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Parent Case

Dupixent Lawsuit Lawsuit

Dupixent (dupilumab) is a monoclonal antibody biologic drug developed by Regeneron Pharmaceuticals and marketed jointly with Sanofi-Aventis and its subsidiary Genzyme Corporation. The FDA first approved Dupixent in March 2017 for adults with moderate-to-severe atopic dermatitis (eczema) who had not responded adequately to topical treatments. Subsequent FDA approvals expanded its use to moderate-to-severe asthma in October 2018, chronic rhinosinusitis with nasal polyps (CRSwNP) in June 2019, eosinophilic esophagitis (EoE) in May 2022, prurigo nodularis in September 2022, and chronic obstructive pulmonary disease (COPD) with an eosinophilic phenotype in September 2024. With more than 37 million prescriptions dispensed globally, Dupixent became one of the best-selling biologic drugs in the world, generating over $13 billion in annual revenue for its manufacturers. The drug works by inhibiting interleukin-4 (IL-4) and interleukin-13 (IL-13) signaling — two cytokines that drive type 2 inflammation. While this mechanism was considered targeted and relatively safe, post-market surveillance and independent research have identified a concerning association between Dupixent use and cutaneous T-cell lymphoma (CTCL). CTCL is a rare cancer in which T-cells become malignant and attack the skin, often presenting initially as rashes or patches that can be mistaken for eczema. The critical concern is that symptoms of CTCL closely mimic the very conditions Dupixent is prescribed to treat, potentially masking the cancer and delaying diagnosis. A peer-reviewed study analyzing 19,612 patients found that Dupixent users faced a 4.5 times higher risk of developing CTCL compared to patients not using the drug. The FDA placed Dupixent on its drug safety watchlist in March 2025 and escalated to a formal investigation in September 2025 after receiving over 300 adverse event reports related to lymphoma and blood cancers. As of February 2026, individual lawsuits are being filed against Regeneron Pharmaceuticals, Sanofi-Aventis, and Genzyme Corporation. Plaintiffs allege that the manufacturers knew or should have known about the cancer risk and failed to adequately warn patients and prescribing physicians. Specific allegations include failure to warn, defective design of the drug's labeling, negligence in post-market surveillance, and fraudulent concealment of safety data. A wrongful death suit was filed in Tennessee in October 2025 on behalf of Chandra Richardson. Additional cases have been filed in Florida (January 2026) and Illinois (December 2025). Legal experts anticipate that the Judicial Panel on Multidistrict Litigation will consolidate federal cases into an MDL within the next 12 months.

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